......continued
But (some will argue) conditions like migraine, fibromyalgia and nerve pain have nothing to do with inflammation. That's true, they don't have the kind of inflammation you can see under a microscope. But what you do see in those conditions are lots of pro-inflammatory cytokines - and that's a problem. (And in fibromyalgia it's probably the problem.)
Cytokines are chemical messengers, and pro-inflammatory cytokines are (not surprisingly) messengers that spread inflammation. So if you have too many pro-inflammatory cytokines (like in fibromyalgia) then you have inflammation - or at least a form of inflammation.
To put things in perspective, if inflammation has 100 (hypothetical) steps, then NF-kB activation is step 1, and pro-inflammatory cytokine production is step 2. It's not until around step 68 that you can see inflammation under a microscope, and aspirin works at about step 43.
The point, of course, is that if step 2 is the problem, you'll never solve it by shooting at step 43. But you can take care of problems at steps 2 through infinity by turning down the 'master switch' - NF-kB. That's what we're trying to do with Banjo.
So if Banjo stops inflammation at step 1 - why doesn't it work right away?
Well, sometimes it does - but inflammation is a process. Think of an assembly line. If you barricade the loading dock (block step 1) finished product (pain) can still be assembled - for a while. But eventually your body is going to run low on the 'raw materials' needed for pain. Production slows, then ceases altogether. That's the goal.